作者: T.A. Sears
DOI: 10.1016/S0079-6123(08)60627-5
关键词:
摘要: Publisher Summary In spontaneous respiration, thoracic respiratory motoneurones show slow, rhythmic fluctuations of their membrane potentials. These slow potentials are named “central drive (CRPDs).” The CRDPs consist phases depolarization and hyperpolarization in alternating succession, the phase being a synaptically induced inhibition. phased inhibition is considerable functional significance because it provides means by which central nervous mechanism respiration exerts control over segmental proprioceptive reflexes muscles. If abolished hyperventilation or signalization, may be demonstrated that when chest inflated, activity evoked expiratory muscles operation spinal reflex. Any mechanical factor, causes shortening extra fusal fibres to lag on intrafusal fibres, such as an increased resistance air flow from lungs, leads discharge muscle spindles with consequence intercostal subjected excitatory drive.