作者: Jacob Fine
DOI: 10.1001/ARCHSURG.1968.01330200001001
关键词:
摘要: IN THIS paper I shall present new evidence in the support of hypothesis that a bacterial factor is responsible for refractory state traumatic shock. This warrants reshaping as follows: shock result ischemic damage to reticuloendothelial system (RES) liver and spleen, this injury allows neurotoxin origin produce fatal collapse peripheral circulation. The will include data showing perfusion arterial blood less than one hour through freshly excised donor spleen eliminates toxin thereby effects recovery from Report Cases begin with recent clinical experience. A man 70 left hospital good order week after surgical repair an obstructing duodenal ulcer. Five days later, when he returned inspection his wound,