Computing Endolymph Hydrodynamics During Head Impulse Test on Normal and Hydropic Vestibular Labyrinth Models.

摘要: Hypothesis: Build a biologic geometry based computational model to test the hypothesis that, in some circumstances, endolymphatic hydrops can mechanically cause enhanced eye velocity responses during clinical conditions of head impulse test. Background: Some recent and experimental findings had suggested that measured with video could not only be caused by recording artifacts or central disfunction but also directly mechanical effect on horizontal semicircular canal receptor. Methods: Data from was computed three biologic-based models governed Navier-Stokes equations; six impulses incrementally increasing peak were each one different geometric models, depending absence, utricular hydrops. Results: For all an increased pressure at ampullar region both models. The mean aVOR gain 1.01 ± 0.008 for no-hydrops model, 1.14 0.010 was, 1.10 0.007 model. Conclusion: results physical computation support-the conditions, which are affecting moderate dilatations, output-by will factor values over 1.1 right side.