作者: Yuji Toiyama , Yasuhiro Inoue , Junichiro Hiro , Eiki Ojima , Hideki Watanabe
DOI: 10.1007/S00280-006-0327-1
关键词: Cell cycle 、 Gastrointestinal cancer 、 DNA repair 、 ERCC1 、 Radiation-Sensitizing Agents 、 Cell culture 、 VEGF receptors 、 Biology 、 Paclitaxel 、 Cancer research 、 Internal medicine 、 Endocrinology
摘要: Aim This study was performed to investigate the range of optimal concentration and mechanisms paclitaxel (PXL) radio-enhancement in gastrointestinal cancer cell lines HT29 MKN45.