Treatment of tumors with autologous LAK cells, interleukin-2 and an ornithine decarboxylase inhibitor
作者： Terry L. Bowlin , Sai P. Sunkara
DOI:
关键词: Lymphokine-activated killer cell 、 Molecular biology 、 Interleukin 2 、 Chemistry 、 Neoplastic disease 、 Ornithine Decarboxylase Inhibitor 、 Tumor Load
摘要: Ornithine decarboxylase inhibitors when administered in conjunction with autologous LAK cells and interleukin-2 provide for an enhanced treatment of neoplastic disease states. This enhancement is provided by the ability ornithine inhibitor to reduce tumor load enhance production T helper cells.
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