Inherited mouse mutations as models of human adnexal, cornification, and papulosquamous dermatoses
作者： John P. Sundberg , Wesley G. Beamer , Leonard D. Shultz , Robert W. Dunstan
DOI: 10.1111/1523-1747.EP12505816
关键词: Ichthyosis 、 Hairless 、 Sebaceous gland 、 Pathology 、 Animal model 、 Psoriasis 、 Hyperkeratosis 、 Hypoplasia 、 Abnormality 、 Medicine
摘要: Nearly 100 mouse mutations have been described as causing some type of abnormality the skin or hair. As only a few these studied in detail, they remain an untapped resource for furthering knowledge basic cutaneous physiology and understanding pathophysiology analogous diseases humans. Several diverse murine are discussed. These include "asebia," mildly hyperkeratotic disorder with sebaceous gland hypoplasia; "ichthyosis," example abnormal hair growth associated hyperkeratosis; "rhino" "hairless," two related examples congenital follicular malformations; "flaky skin", potential animal model eruptive psoriasis.
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