Plasminogen-activator inhibitor type 1 and coronary artery disease.
作者： Hans P. Kohler , Peter J. Grant
DOI: 10.1056/NEJM200006153422406
关键词: Internal medicine 、 Myocardial infarction 、 Plasminogen activator inhibitor-1 、 Cardiology 、 Fibrinolysis 、 Endocrinology 、 Pathogenesis 、 Medicine 、 Coronary artery disease 、 Vascular disease 、 Thrombus 、 Atheroma
摘要: The development of coronary artery disease, and specifically myocardial infarction, involves hyperplasia arterial smooth muscle, the fatty streaks, atheroma formation, plaque rupture, ultimately thrombus formation vessel occlusion.1 These changes are in part genetically determined, as demonstrated by fact that risk infarction persons who have a first-degree relative with is seven times do not.2,3 This finding often used to argue disease has genetic basis, but extent which shared environment contributes must also . . .
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