作者: Ralf A. Linker , De-Hyung Lee , Sarah Ryan , Anne M. van Dam , Rebecca Conrad
DOI: 10.1093/BRAIN/AWQ386
关键词: Protein nitrosylation 、 Multiple sclerosis 、 Experimental autoimmune encephalomyelitis 、 Neuroinflammation 、 Encephalomyelitis 、 Myelin oligodendrocyte glycoprotein 、 Neuroprotection 、 Myelin 、 Pharmacology 、 Immunology 、 Biology
摘要: Inflammation and oxidative stress are thought to promote tissue damage in multiple sclerosis. Thus, novel therapeutics enhancing cellular resistance to free radicals could prove useful …