The Relevance of the TGF-β Paradox to EMT-MET Programs
作者： Chevaun D. Morrison , Jenny G. Parvani , William P. Schiemann
DOI: 10.1016/J.CANLET.2013.02.048
关键词: Cell 、 Epithelial–mesenchymal transition 、 Biology 、 Cell migration 、 Cancer research 、 Transforming growth factor 、 Transforming growth factor beta 、 Carcinogenesis 、 Immunology 、 microRNA 、 Metastasis
摘要: … of miR-200 family members, which function in suppressing the expression of the EMT transcription factors, ZEB1 and ZEB2 (SIP1) [31], [32]. During EMT and metastasis stimulated by …
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