Increased inactivation of nitric oxide is involved in impaired coronary flow reserve in heart failure.
作者： Ryo Nakamura , Kensuke Egashira , Kenichi Arimura , Youji Machida , Tomomi Ide
DOI: 10.1152/AJPHEART.2001.281.6.H2619
关键词: Circulatory system 、 Nitric oxide 、 Medicine 、 Vasodilation 、 Blood vessel 、 Heart failure 、 Endothelial dysfunction 、 Coronary flow reserve 、 Endocrinology 、 Internal medicine 、 Endothelial stem cell
摘要: Recent evidence suggests that increased inactivation of endothelium-derived nitric oxide (NO) by oxygen free radical (OFR) formation is involved in the pathogenesis endothelial dysfunction he...
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