作者: Tomoka Takatani , Kyoko Takahashi , Chengshi Jin , Takahisa Matsuda , Xinyao Cheng
DOI: 10.1097/00005344-200406000-00008
关键词: Medicine 、 Minoxidil 、 Neonatal rat 、 Ischemia 、 Simulated ischemia 、 Internal medicine 、 Endocrinology 、 Apoptosis 、 Necrosis
摘要: Abstract:The effects of minoxidil (a mitochondrial K+ATP channel opener) on ischemia-induced necrosis and apoptosis were examined using a cardiomyocyte model simulated ischemia, since mito-chondrial openers have been suggested to be involved in the mechanisms cardioprotective act