Transmethylation hypothesis of schizophrenia: methionine and nicotinic acid.
作者： J.N. Nestoros , T.A. Ban , H.E. Lehmann
DOI: 10.1159/000468311
关键词: Exacerbation 、 Monoamine oxidase inhibitor 、 Methionine 、 Transmethylation 、 Schizophrenia 、 Pharmacology 、 Psychopathology 、 Medicine 、 Nicotinic agonist
摘要: The transmethylation hypothesis of schizophrenia was reviewed with considerations that large doses methionine when combined a monoamine oxidase inhibitor lead to exacerbation psychotic symptoms in significant percentage chronic schizophrenic patients. It noted nicotinic acid the dosage 3,000 mg/day can neither prevent nor counteract psychopathology thus induced.
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