作者: Harriet Wikman , Päivi Piirilä , Christina Rosenberg , Ritva Luukkonen , Katja Kääriä
DOI: 10.1097/00008571-200204000-00007
关键词: N-acetyltransferase 、 Risk factor 、 Genotype 、 Asthma 、 Immunology 、 Gene 、 Biology 、 Arylamine N-acetyltransferase 、 Lung disease 、 Glutathione
摘要: We observed previously that polymorphisms in glutathione S-transferase (GST) genes modified allergic responses to diisocyanate exposure. Here, we extended the study examine possible role of N-acetyltransferase (NAT) genotypes development diisocyanate-induced ill effects, both separa