Human Common Fragile Site FRA16D Flexibility Peak Is Not a Strong Mitotic Recombination Hotspot in Saccharomyces cerevisiae
作者： Katina G. Kapellas
DOI:
关键词: DNA 、 Chromosomal fragile site 、 Genetics 、 Saccharomyces cerevisiae 、 Loss of heterozygosity 、 Biology 、 Mitotic crossover
摘要:
参考文章(57)
William Joseph Fitzsimmons, Human common fragile sites, identification, and analysis of breaks using saccharomyces cerevisiae ,(2013)
Haihua Zhang, Catherine H. Freudenreich, An AT-rich Sequence in Human Common Fragile Site FRA16D Causes Fork Stalling and Chromosome Breakage in S. cerevisiae Molecular Cell. ,vol. 27, pp. 367- 379 ,(2007) , 10.1016/J.MOLCEL.2007.06.012
Francene J. Lemoine, Natasha P. Degtyareva, Kirill Lobachev, Thomas D. Petes, Chromosomal Translocations in Yeast Induced by Low Levels of DNA Polymerase: A Model for Chromosome Fragile Sites Cell. ,vol. 120, pp. 587- 598 ,(2005) , 10.1016/J.CELL.2004.12.039
Anne M. Casper, Paul Nghiem, Martin F. Arlt, Thomas W. Glover, ATR Regulates Fragile Site Stability Cell. ,vol. 111, pp. 779- 789 ,(2002) , 10.1016/S0092-8674(02)01113-3
Marion Cooley, Nawin C. Mishra, Genetic analysis of the in vivo role of DNA polymerases in Saccharomyces cerevisiae. Current Genetics. ,vol. 38, pp. 256- 263 ,(2000) , 10.1007/S002940000163
Thomas W. Glover, Carol Berger, Jane Coyle, Barbara Echo, DNA polymerase ? inhibition by aphidicolin induces gaps and breaks at common fragile sites in human chromosomes Human Genetics. ,vol. 67, pp. 136- 142 ,(1984) , 10.1007/BF00272988
Efrat Ozeri-Galai, Assaf C. Bester, Batsheva Kerem, The complex basis underlying common fragile site instability in cancer. Trends in Genetics. ,vol. 28, pp. 295- 302 ,(2012) , 10.1016/J.TIG.2012.02.006
R. S. Cha, ATR Homolog Mec1 Promotes Fork Progression, Thus Averting Breaks in Replication Slow Zones Science. ,vol. 297, pp. 602- 606 ,(2002) , 10.1126/SCIENCE.1071398
Benoît Le Tallec, Gaël Armel Millot, Marion Esther Blin, Olivier Brison, Bernard Dutrillaux, Michelle Debatisse, Common Fragile Site Profiling in Epithelial and Erythroid Cells Reveals that Most Recurrent Cancer Deletions Lie in Fragile Sites Hosting Large Genes Cell Reports. ,vol. 4, pp. 420- 428 ,(2013) , 10.1016/J.CELREP.2013.07.003
Susan W. Liebman, Irina L. Derkatch, The yeast [PSI+] prion: making sense of nonsense. Journal of Biological Chemistry. ,vol. 274, pp. 1181- 1184 ,(1999) , 10.1074/JBC.274.3.1181