Selective neuronal nitric oxide synthase inhibition blocks furosemide-stimulated renin secretion in vivo
作者： W. H. Beierwaltes
DOI: 10.1152/AJPRENAL.1995.269.1.F134
关键词: Renin–angiotensin system 、 In vivo 、 Pharmacology 、 Endocrinology 、 Nitric oxide synthase 、 Nitric oxide 、 Secretion 、 Chemistry 、 Macula densa 、 Gene isoform 、 Kidney 、 Internal medicine
摘要: The macula densa is a regulatory site for renin. It contains exclusively the neuronal isoform of nitric oxide synthase (NOS), suggesting NO could stimulate renin secretion through ...
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