A reappraisal of the evidence that rheumatoid arthritis and several other idiopathic diseases are slow bacterial infections.

作者: G A Rook , P M Lydyard , J L Stanford

DOI: 10.1136/ARD.52.SUPPL_1.S30

关键词: ImmunologyCellular immunityInflammatory bowel diseaseMedicineSystemic diseaseRheumatoid arthritisAutoimmune diseaseImmune systemReactive arthritisAutoimmunityGeneral Biochemistry, Genetics and Molecular BiologyImmunology and AllergyRheumatology

摘要:

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