Ultra-endurance exercise induces stress and inflammation and affects circulating hematopoietic progenitor cell function.

摘要: Although amateur sports have become increasingly competitive within recent decades, there are as yet few studies on the possible health risks for athletes. This study aims to determine impact of ultra-endurance exercise-induced stress number and function circulating hematopoietic progenitor cells (CPCs) hematological, inflammatory, clinical, metabolic, parameters in moderately trained Following exercise, were significant increases leukocytes, platelets, interleukin-6, fibrinogen, tissue enzymes, blood lactate, serum cortisol, matrix metalloproteinase-9. Ultra-endurance exercise did not influence CPCs but resulted a highly decline CPC functionality after competition. Furthermore, Epstein-Barr virus was seen be reactivated one seven The link between is supported by negative correlation cortisol function. We conclude that induces metabolic an inflammatory response affects only mature also immature stem cell fraction, which make up immune system provide regeneration.