作者: Stephen M. Oppenheimer , Vladimir C. Hachinski
DOI: 10.1016/S0733-8619(18)30239-1
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摘要: Stroke, whether ischemic or hemorrhagic, induces cardiac damage by nonischemic mechanisms. The evidence derives from autopsy studies and investigation of ECG, enzyme changes, plasma catecholamine changes after stroke. Increased sympathoadrenal tone, resulting to cortical areas involved in autonomic control is the likely cause. Recent experimental indicates that insular cortex plays a principal role stroke-related damage.