Intestinal ischemia and shock factors.

摘要: Perfusion pressure reduction to the small intestinal vascular bed for 2 hours followed by characteristic villous damage and a general cardiovascular derangement. In an extensive series of experiments on cats it was demonstrated that derangement could not be ascribed any pooling blood and/or fluid in intestine. Cardiotoxic material has been cat venous vivo vitro using working rat hearts isolated rabbit papillary muscles. A relationship between extent appearance cardiotoxic found. The seems consist at least two heat stable fractions. One is water soluble with molecular mass 500 1,000 daltons other lipid unknown mass.