作者: Min Lou , Chao-hui Jing , Magdy H. Selim , Louis R. Caplan , Mei-ping Ding
DOI: 10.1016/J.JNS.2008.09.032
关键词:
摘要: Delayed post-anoxic encephalopathy has rarely been reported, and most commonly associated with carbon monoxide poisoning. The underlying pathophysiological mechanism is unknown. We describe a patient delayed neurologic decline after good initial recovery from hypoxic-ischemic event precipitated by acute blood loss. MRI revealed early globus pallidus necrosis followed substantia nigra damage leukoencephalopathy. suggest that anemia might play role in this pattern of brain injury.