Delayed Release of Intracellular Microcystin Following Partial Oxidation of Cultured and Naturally Occurring Cyanobacteria.

摘要: Oxidation processes can provide an effective barrier to eliminate cyanotoxins by damaging cyanobacteria cell membranes, releasing intracellular cyanotoxins, and subsequently oxidizing these toxins (now in extracellular form) based on published reaction kinetics. In this work, cells from two natural blooms (from the United States Canada) a laboratory-cultured Microcystis aeruginosa strain were treated with chlorine, monochloramine, chlorine dioxide, ozone, potassium permanganate. The release of microcystin was measured immediately after oxidation (t ≤ 20 min), following oxidant residual quenching (stagnation times = 96 or 168 h). Oxidant exposures (CT) determined resulting complete (21 mg-min/L), chloramine (72 dioxide (58 ozone (4.1 permanganate (391 mg-min/L). Required using indigenous greater than lab-cultured Microcystis. Following partial (oxidant CT values cited above), additional dissolved organic carbon (DOC) released while samples remained stagnant absence (>96 h quenching). delayed partially oxidized has implications for drinking water treatment as may be retained filter surface solids continue slowly other metabolites into finished water.