作者: Franz H. Messerli , Leszek Michalewicz
DOI: 10.1007/978-1-4615-5385-4_28
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摘要: Left ventricular hypertrophy (LVH) has been identified as one of the strongest pressure-independent risk factors for sudden death, acute myocardial infarction, congestive heart failure and other cardiovascular morbidity mortality. [1,2,3] These findings clearly abolish concept LVH being a benign adaptive process serving to compensate increased hemodynamic burden. Fig. 1. Based on these epidemiologic observations, patophysiologic or electrophysiologic chain evidence linking hypertension, LVH, death put forward. Although it is unclear whether reduction in confers benefit over above lowering blood pressure alone, effects various antihypertensive agents have come under scrutiny.