作者: SEYMOUR REICHLIN
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摘要: Electrolytic lesions of the hypothalamus were produced in 73 male rats. When compared with 64-sham-operated controls, certain rats had thyroid inhibition (decreased I131 uptake and release rate), persistent hyperthormia, diabetes insipidus, tosticular atrophy growth disturbance. Functional neuroanatomical studies indicated that dysfunction, hyperthermia, insipidus testicular all independent consequences localized brain injury. A temperature regulating center was to pre-optic area; this partially overlapped TSH area which lies between paraventricular nucleus median eminence. Gonad caused by infundibular damage. As measured rate increase body length (nose-tail) tibial at death, significantly impaired hypothalamic lesions. No specific neural localization lesion responsible for poor could be made but it noted most striking effect...