The role of TNF-α induced protein 1 in the activation of pro-apoptotic proteins.
作者： Serge Dibart , Xiaoren Tang , Mingfang Su , Bushra Aljahdali , Sean Lee
DOI: 10.1007/S13577-021-00529-9
关键词:
摘要: It is known that Porphyromonas gingivalis/lipopolysaccharide (P. gingivalis/LPS) induces inflammatory diseases via TNF-α-mediated transcription factors. Our recent data shows TNFAIP1 (TNF-α induced protein 1) related to TNF-α. However, little regarding how involved in the TNF-α-dependent pathway. We therefore focused on biological function of and examined mediates TNF-α other genes. found was upregulated peaks before upregulation apoptotic genes such as Bad, Bcl-x, Caspase 3, Catalase, Claspin, Cytochromic, Ho-1/HMOX1/HSP32, or MCI-1 our time course with TNFAIP1-treated cells. findings here may serve foundation for future studies linking regulation intervention disease.
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