作者: Sameh K. Wasseff , Steven S. Scherer
DOI: 10.1016/J.JNEUROIM.2014.02.005
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摘要: Abstract We investigated whether microglia form gap junctions with themselves, or astrocytes, oligodendrocytes, neurons in vivo normal mouse brains, and pathological conditions that induce microglial activation — brain injury a model of Alzheimer's disease. Although are close physical proximity to glia neurons, they do not functional under these conditions.