作者: Shameem Mahmood , Janet A. Gilbertson , Nigel Rendell , Carol J. Whelan , Helen J. Lachmann
DOI: 10.1182/BLOOD-2014-06-580720
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摘要: To the editor: Amyloidosis is a heterogeneous group of diseases, in which amyloidogenic precursor proteins misfold and adopt β-pleated sheet conformation.[1][1] Several can form amyloid fibrils vivo including transthyretin,[2][2] apolipoprotein A-I A-II, lysozyme, fibrinogen,