Endogenous mediators in sepsis and septic shock.
作者： A. Beishuizen , I. Vermes , C. Haanen
DOI: 10.1016/S0065-2423(08)60206-1
关键词:
摘要: Publisher Summary The chapter introduces the pathogenesis of sepsis and various definitions sepsis. status cytokines are discussed wherein proinflammatory (TNF, interleukin-1, interleukin-8); anti-inflammatory (interleukin-6 interleukin- 10); soluble TNF IL-1 receptors, IL-2 receptor antagonists; heat shock proteins (ischemia-reperfusion, ARDS, multiple organ dysfunction syndrome) described. also discusses two vasoactive agents, endothelin, nitric oxide: NO vascular hyporeactivity in sepsis, myocardial contractility experimental septic shock, clinical Hemostasis is role plasma cascade systems (the coagulation system, contact fibrinolytic complement system) endothelium (endothelial cells as regulators therapeutic approaches to attenuate sepsis) Various hormonal regulation—namely, stress hormones (hypothalamic-pituitary-adrenal axis, catecholamines other hormones); fluid electrolyte homeostasis (vasopressin, renin-angiotensin-aldosterone atrial natriuretic peptide); pituitary-thyroid axis; reproductive axis chapter. interplay mediators presented.
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sci-hub.se 参考文章(465)
I. Engelberts, W. A. Buurman, A. Moller, G. J. M. Schoen, C. J. Van Der Linden, Evaluation of measurement of human TNF in plasma by ELISA. Lymphokine and cytokine research. ,vol. 10, pp. 69- 76 ,(1991)
G E Grau, P Suter, J M Dayer, P Roux-Lombard, E Girardin, H Gallati, Imbalance between tumour necrosis factor-alpha and soluble TNF receptor concentrations in severe meningococcaemia. The J5 Study Group. Immunology. ,vol. 76, pp. 20- 23 ,(1992)
RA DeLa Cadena, AF Suffredini, JD Page, RA Pixley, N Kaufman, JE Parrillo, RW Colman, Activation of the kallikrein-kinin system after endotoxin administration to normal human volunteers Blood. ,vol. 81, pp. 3313- 3317 ,(1993) , 10.1182/BLOOD.V81.12.3313.3313
TM Carlos, BR Schwartz, NL Kovach, E Yee, M Rosa, L Osborn, G Chi-Rosso, B Newman, R Lobb, M] Rosso M$[corrected to Rosa, Vascular cell adhesion molecule-1 mediates lymphocyte adherence to cytokine-activated cultured human endothelial cells. Blood. ,vol. 76, pp. 965- 970 ,(1990) , 10.1182/BLOOD.V76.5.965.965
McMillen Ma, Cunningham Me, D'Aiuto Ml, Pleban We, Huribal M, Endothelin-1 and prostaglandin E2 levels increase in patients with burns. Journal of The American College of Surgeons. ,vol. 180, pp. 318- 322 ,(1995)
Dinarello Ca, Revhaug A, Michie Hr, Manogue Kr, Sherman Ml, Wolff Sm, O'Dwyer St, Cerami A, Arthur K, Spriggs Dr, Tumor necrosis factor and endotoxin induce similar metabolic responses in human beings. Surgery. ,vol. 104, pp. 280- 286 ,(1988)
JH Nuijens, CC Huijbregts, AJ Eerenberg-Belmer, JJ Abbink, RJ Strack van Schijndel, RJ Felt-Bersma, LG Thijs, CE Hack, Quantification of plasma factor XIIa-Cl(-)-inhibitor and kallikrein-Cl(-)-inhibitor complexes in sepsis. Blood. ,vol. 72, pp. 1841- 1848 ,(1988) , 10.1182/BLOOD.V72.6.1841.1841
Bart J Biemond, Marcel Levi, Hugo ten Cate, Howard R Soule, Lori D Morris, David L Foster, Cheryl A Bogowitz, Tam van der Poll, Harry R Büller, Jan Wouter ten cate, Complete inhibition of endotoxin-induced coagulation activation in chimpanzees with a monoclonal Fab fragment against factor VII/VIIa. Thrombosis and Haemostasis. ,vol. 73, pp. 223- 230 ,(1995) , 10.1055/S-0038-1653755
Lerner B. Hinshaw, Sepsis/septic shock: participation of the microcirculation: an abbreviated review. Critical Care Medicine. ,vol. 24, pp. 1072- 1078 ,(1996) , 10.1097/00003246-199606000-00031
Yoshida M, Endo S, Suzuki M, Inada K, Hoshi S, Kuwata Y, Inoue Y, Yamashita H, Two types of septic shock classified by the plasma levels of cytokines and endotoxin. Circulatory shock. ,vol. 38, pp. 264- 274 ,(1992)