作者: S. Engel , H. D. Wehner , R. Meyermann
DOI: 10.1007/978-3-7091-9465-2_16
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摘要: The loss of neurons after severe closed head injury is not only a consequence the primary impact but also secondary damage mechanisms. Among cell population central nervous system microglia surely candidate to influence mechanisms by releasing cytotoxic cytokines [1]. About microglial reaction in traumatic brain (TBI), however, no data are available. In contrast experiments using stab wound coverings CNS TBI still intact.