Genetic Dissection of Blackleg Resistance Loci in Rapeseed (Brassica napus L.)

作者: Harsh Raman , Rosy Raman , Nick Lark

DOI: 10.5772/53611

关键词:

摘要: Blackleg disease caused by the heterothallic ascomycete fungus Leptosphaeria maculans (Desm.) Ces. et de Not. (anamorph: Phoma lingam Tode ex Fr.), is major of Brassica crops such as turnip rape (Brassica rapa L. syn. B. campestris; 2n = 2x 20, genome AA), cabbage (B. oleracea L.; 18, CC), rapeseed (syn. canola or oilseed napus 4x 38, AACC), and juncea (Indian brown mustard; 36, AABB) grown in temperate regions world. It was recorded for first time on stems red [1]. originated a result natural interspecific hybridization doubling between monogenomic diploid species, oleracea, southern Europe approximately 10,000–100,000 years ago [2, 3]. However, it selected an crop only 300-500 [4, 5]. originally evolved spring semi-winter type under Mediterranean climates, spread rapidly from to northern after development winter varieties [6]. Both types are affected blackleg disease, particularly Australia, North America. Currently world’s third most important crop, area over 23 million hectares produce almost 53.3 tonnes annually [7]. Increase production has been attributed release high yielding superior including hybrids having traits oil content, improved protein quality herbicide resistance better management.

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