The effect of cold air inhalation on again pectoris and myocardial oxygen supply.

摘要: Inhalation of cold air (-20 degrees C) for four minutes provoked angina pectoris in 17 coronary disease patients at rest and seven the while they were paced a heart rate level which was subanginal room temperature. The did not increase myocardial O2 consumption significantly, accompanying changes systemic hemodynamic factors known to influence minor. Coronary blood flow determined by xenon clearance method change significantly. In 18 patients, inhalation 1 1/2 caused no detectable constriction arteries visualized arteriographically. We conclude that induced breathing cannot be explained satisfactorily concurrent work consumption. Although neither large artery nor generalized arteriole seem involved, some other specific effect on vasomotion, perhaps affecting collaterals or distribution, is suspected.