摘要: In the world of modern medicine it is rare that understanding a previously described clinical disease so completely revolutionized textbooks must be rewritten. This, however, has been outcome isolation ofHelicobacter pylori from human stomach. Only 15 years after its initial isolation, this bacterium proven to etiological agent acute on chronic gastritis, and predisposing factor in peptic ulcer disease, gastric carcinoma B cell mucosa-associated lymphoid tissue (MALT) lymphoma (Graham et al. 1992; IARC 1994; Marshall 1985; Parsonnet 1994).
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