作者: M. Frotscher , M. Schlander , S. Hoyer
DOI: 10.1016/0304-3940(88)90687-8
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摘要: Glutamate decarboxylase (GAD)-immunoreactive, supposedly GABAergic inhibitory, neurons in various fields of the rat hippocampus and pyramidal cells area CA1 were quantified 1 week after transient cerebral ischemia by 4-vessel occlusion. Whereas number Toluidine blue-stained semithin sections found reduced 50% when compared with controls there was no loss GAD-immunoreactive vibratome proper fascia dentata. These data suggest that hippocampal are more resistant to than cells.