Neural control of the renal circulation

摘要: Abstract All elements of the renal vasculature receive a predominant noradrenergic innervation, with norepinephrine as functional neurotransmitter. Basal levels efferent sympathetic nerve activity to are low. In basal state, decreases (renal denervation, arterial and cardiac baroreceptor stimulation) in already low level do not increase blood flow or decrease vascular resistance. A variety afferent reflex inputs (somatic, visceral, environmental stress) produce increases which The responses activation nerves mediated by cc.,-adrenoceptors located on elements. ultimate response is influenced participation paracrine factors (angiotensin II, prostaglandins), whose secretion may be activity.