作者: L.M Agius
DOI: 10.1016/S0306-9877(03)00176-2
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摘要: Abstract Amyloid fibrillogenesis as a process of interactive molecular processes deposition in Alzheimer’s disease might function phenomenon that transforms intracellular amyloid segregation to state equilibration with extracellular deposition. β-Amyloidosis dynamically implicate loss viability vascular tunica media myofibers strict reflection neurons such an overall system between and fibrillogenesis. In terms beyond simple concepts biophysical equilibration, β-amyloid constitute congophilic angiopathy pathobiologic index activity the Alzheimer process; correlate would perhaps involve quantitative qualitatively characterize series reactions microenvironment.