作者: Steven A Barker , G.F Carl , John A Monti
DOI: 10.1016/0306-9877(80)90087-0
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摘要: Abstract A biochemical hypothesis concerning the etiology of schizophrenia is presented. This postulates presence a genetically determined lesion in disposition one-carbon units leading to elevated levels formaldehyde, i.e. hyperformaldehydism. The relationships between this and existing major hypotheses schizoprenia regarding dopamine transmethylation are discussed.