作者: Sunil S. Metkar , Christopher J. Froelich
DOI: 10.1182/BLOOD-2004-03-0888
关键词:
摘要: Wagner et al[1][1] and Hochegger al[2][2] reported that human polymorphonuclear neutrophils (PMNs) express granzyme A (GzmA), B (GzmB), perforin (PFN), postulating a role for these granule proteins in PMN-mediated antibody-dependent cellular cytotoxicity. Sayers al[3][3] had