作者: Murali-Mohan Ayyanath , Cynthia D. Scott-Dupree , G. Christopher Cutler
DOI: 10.1016/J.CHEMOSPHERE.2015.01.061
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摘要: Abstract Insect reproduction can be stimulated by exposure to sublethal doses of insecticide that kill the same insects at high doses. This bi-phasic dose response a stressor is known as hormesis and has been demonstrated with many different insect–insecticide models. The specific mechanisms increased in following pesticide are unknown, but may related juvenile hormone (JH), which major role regulation metamorphosis reproductive development insects. We tested hypothesis concentrations precocene, an antagonist JH, would not result outputs green peach aphid, Myzus persicae, neurotoxic insecticides. also measured JH titers expression various developmental (FPPS I), stress (Hsp60), dispersal (OSD, TOL ANT) genes aphids precocene treatments. found when aphid nymphs were treated certain 1.5- 2-fold stimulation occurred they became adults, this effect subsided generation. Precocene treatments resulted no measurable effects on levels subsequent reproducing adults. Although we detected gene some (e.g. 100- 300-fold genes), there clear relationships between responses for given treatment.