In BCR-ABL-positive cells, STAT-5 tyrosine-phosphorylation integrates signals induced by imatinib mesylate and Ara-C.
作者： T Kindler , F Breitenbuecher , S Kasper , T Stevens , B Carius
关键词:
摘要: In BCR-ABL-positive cells, STAT-5 tyrosine-phosphorylation integrates signals induced by imatinib mesylate and Ara-C
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S. Clark, J McLaughlin, M Timmons, A. Pendergast, Y Ben-Neriah, L. Dow, W Crist, G Rovera, S. Smith, O. Witte, Expression of a Distinctive BCR-ABL Oncogene in Ph1-positive Acute Lymphocytic Leukemia (ALL) Science. ,vol. 239, pp. 775- 777 ,(1988) , 10.1126/SCIENCE.3422516
Agata Klejman, Steven J Schreiner, Malgorzata Nieborowska-Skorska, Artur Slupianek, Matthew Wilson, Thomas E Smithgall, Tomasz Skorski, The Src family kinase Hck couples BCR/ABL to STAT5 activation in myeloid leukemia cells The EMBO Journal. ,vol. 21, pp. 5766- 5774 ,(2002) , 10.1093/EMBOJ/CDF562
Eric Rhéaume, Luchino Y Cohen, Frank Uhlmann, Claude Lazure, Antoine Alam, Jerard Hurwitz, Rafick‐Pierre Sékaly, François Denis, The large subunit of replication factor C is a substrate for caspase‐3 in vitro and is cleaved by a caspase‐3‐like protease during Fas‐mediated apoptosis The EMBO Journal. ,vol. 16, pp. 6346- 6354 ,(1997) , 10.1093/EMBOJ/16.21.6346
JANET D. ROWLEY, Letter: A new consistent chromosomal abnormality in chronic myelogenous leukaemia identified by quinacrine fluorescence and Giemsa staining. Nature. ,vol. 243, pp. 290- 293 ,(1973) , 10.1038/243290A0
Daniel E Epner, H Phillip Koeffler, Molecular Genetic Advances in Chronic Myelogenous Leukemia Annals of Internal Medicine. ,vol. 113, pp. 3- 6 ,(1990) , 10.7326/0003-4819-113-1-3
Brian J. Druker, Shu Tamura, Elisabeth Buchdunger, Sayuri Ohno, Gerald M. Segal, Shane Fanning, Jürg Zimmermann, Nicholas B. Lydon, Effects of a selective inhibitor of the Abl tyrosine kinase on the growth of Bcr-Abl positive cells. Nature Medicine. ,vol. 2, pp. 561- 566 ,(1996) , 10.1038/NM0596-561
Kiyotsugu Yoshida, Surender Kharbanda, Donald Kufe, None, Functional interaction between SHPTP1 and the Lyn tyrosine kinase in the apoptotic response to DNA damage. Journal of Biological Chemistry. ,vol. 274, pp. 34663- 34668 ,(1999) , 10.1074/JBC.274.49.34663
Merav Socolovsky, Amy E.J Fallon, Stream Wang, Carlo Brugnara, Harvey F Lodish, Fetal Anemia and Apoptosis of Red Cell Progenitors in Stat5a−/−5b−/− Mice: A Direct Role for Stat5 in Bcl-XL Induction Cell. ,vol. 98, pp. 181- 191 ,(1999) , 10.1016/S0092-8674(00)81013-2
A. B. Raitano, J. R. Halpern, T. M. Hambuch, C. L. Sawyers, The Bcr-Abl leukemia oncogene activates Jun kinase and requires Jun for transformation Proceedings of the National Academy of Sciences of the United States of America. ,vol. 92, pp. 11746- 11750 ,(1995) , 10.1073/PNAS.92.25.11746
Jack M. Lionberger, Matthew B. Wilson, Thomas E. Smithgall, Transformation of Myeloid Leukemia Cells to Cytokine Independence by Bcr-Abl Is Suppressed by Kinase-defective Hck Journal of Biological Chemistry. ,vol. 275, pp. 18581- 18585 ,(2000) , 10.1074/JBC.C000126200