作者: Tetsuya Mitsudomi , Yasushi Yatabe
DOI: 10.1111/J.1742-4658.2009.07448.X
关键词: ROR1 、 Gefitinib 、 GRB7 、 Cancer research 、 Receptor tyrosine kinase 、 Biology 、 ERBB3 、 Tyrosine kinase 、 Epidermal growth factor receptor 、 JAK-STAT signaling pathway
摘要: … G598V mutations were found in domain IV. These EGFR mutations occur independently of EGFR vIII and provide an alternative mechanism for EGFR activation in glioblastomas [[8]]. …