A deficiency of coproporphyrinogen III oxidase causes lesion formation in Arabidopsis

摘要: We isolated an Arabidopsis lesion initiation 2 (lin2) mutant, which develops formation on leaves and siliques in a developmentally regulated light-dependent manner. The phenotype of the lin2 plants resulted from single nuclear recessive mutation, LIN2 was by T-DNA tagging approach. encodes coproporphyrinogen III oxidase, key enzyme biosynthetic pathway chlorophyll heme, tetrapyrrole pathway, Arabidopsis. express cytological molecular markers associated with defense responses, usually activated pathogen infection. These results demonstrate that porphyrin impairment is responsible for phenotype, leads to activation Lesion not suppressed, even enhanced when accumulation salicylic acid (SA) prevented expression SA-degrading salicylate hydroxylase (nahG) gene. This suggests triggered determined prior or independently SA but required limit spread lesions plants.