Regulation of smooth muscle proliferative phenotype by heparinoid--matrix interactions.
作者： T. Scott-Burden , F.R. Bühler
DOI: 10.1016/0165-6147(88)90175-7
关键词: Epidermal growth factor 、 Matrix (biology) 、 Heparinoid 、 Cell biology 、 ITGA7 、 Vascular smooth muscle 、 Extracellular 、 Biochemistry 、 Thrombospondin 、 Biology 、 Extracellular matrix
摘要: Abstract The inhibition of vascular smooth muscle cell proliferation by heparin and heparinoid compounds can be correlated with changes in the biosynthesis a number extracellular polypeptides. Furthermore, regulates levels thrombospondin matrix, resulting decreased cellular responsiveness to epidermal growth factor. Tim Scott-Burden Fritz Buhler describe how control phenotypic expression may mediated via heparinoid-extracellular matrix interactions.
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