2-Deoxyadenosine triphosphate restores the contractile function of cardiac myofibril from adult dogs with naturally occurring dilated cardiomyopathy.
作者： Yuanhua Cheng , Kaley A. Hogarth , M. Lynne O'Sullivan , Michael Regnier , W. Glen Pyle
DOI: 10.1152/AJPHEART.00530.2015
关键词: 2'-deoxyadenosine 、 Dilated cardiomyopathy 、 Myosin 、 Endocrinology 、 Cardiac myofibril 、 Internal medicine 、 Chemistry 、 Contraction (grammar) 、 Cardiotonic Agents 、 Myofibril 、 Energy metabolism 、 Anatomy
摘要: … We have previously reported that replacing ATP with 2-deoxyadenosine triphosphate (dATP) as an energy substrate improves contractility of striated muscle by enhancing cross-bridge …
sci-hub.se 参考文章(50)
Ye Luo, Jonathan P. Davis, Svetlana B. Tikunova, Lawrence B. Smillie, Jack A. Rall, Myofibrillar determinants of rate of relaxation in skinned skeletal muscle fibers. Advances in Experimental Medicine and Biology. ,vol. 538, pp. 573- 582 ,(2003) , 10.1007/978-1-4419-9029-7_51
Robert Stehle, Martina Krüger, Gabriele Pfitzer, Does cross-bridge activation determine the time course of myofibrillar relaxation? Advances in Experimental Medicine and Biology. ,vol. 538, pp. 469- 479 ,(2003) , 10.1007/978-1-4419-9029-7_43
Alexandre Fabiato, [31] Computer programs for calculating total from specified free or free from specified total ionic concentrations in aqueous solutions containing multiple metals and ligands☆ Methods in Enzymology. ,vol. 157, pp. 378- 417 ,(1988) , 10.1016/0076-6879(88)57093-3
Kareen L Kreutziger, Nicoletta Piroddi, Beatrice Scellini, Chiara Tesi, Corrado Poggesi, Michael Regnier, None, Thin filament Ca2+ binding properties and regulatory unit interactions alter kinetics of tension development and relaxation in rabbit skeletal muscle. The Journal of Physiology. ,vol. 586, pp. 3683- 3700 ,(2008) , 10.1113/JPHYSIOL.2008.152181
G. Wess, A. Schulze, V. Butz, J. Simak, M. Killich, L.J.M. Keller, J. Maeurer, K. Hartmann, Prevalence of Dilated Cardiomyopathy in Doberman Pinschers in Various Age Groups Journal of Veterinary Internal Medicine. ,vol. 24, pp. 533- 538 ,(2010) , 10.1111/J.1939-1676.2010.0479.X
Shin Kadota, John Carey, Hans Reinecke, James Leggett, Sam Teichman, Michael A Laflamme, Charles E Murry, Michael Regnier, Gregory G Mahairas, None, Ribonucleotide reductase-mediated increase in dATP improves cardiac performance via myosin activation in a large animal model of heart failure European Journal of Heart Failure. ,vol. 17, pp. 772- 781 ,(2015) , 10.1002/EJHF.270
A. TIDHOLM, J. HÄGGSTRÖM, M. BORGARELLI, A. TARDUCCI, Canine Idiopathic Dilated Cardiomyopathy. Part I: Aetiology, Clinical Characteristics, Epidemiology and Pathology Veterinary Journal. ,vol. 162, pp. 92- 107 ,(2001) , 10.1053/TVJL.2001.0571
Kathryn M. Meurs, Sunshine Lahmers, Bruce W. Keene, Stephen N. White, Mark A. Oyama, Evan Mauceli, Kerstin Lindblad-Toh, A splice site mutation in a gene encoding for PDK4, a mitochondrial protein, is associated with the development of dilated cardiomyopathy in the Doberman pinscher Human Genetics. ,vol. 131, pp. 1319- 1325 ,(2012) , 10.1007/S00439-012-1158-2
Steven B. Marston, Pieter P. de Tombe, Troponin phosphorylation and myofilament Ca2+-sensitivity in heart failure: increased or decreased? Journal of Molecular and Cellular Cardiology. ,vol. 45, pp. 603- 607 ,(2008) , 10.1016/J.YJMCC.2008.07.004
Natosha Finley, M.Bret Abbott, Ekram Abusamhadneh, Vadim Gaponenko, Wen-ji Dong, G. Gasmi-Seabrook, Jack W. Howarth, Mark Rance, R.John Solaro, Herbert C. Cheung, Paul R. Rosevear, NMR analysis of cardiac troponin C-troponin I complexes: effects of phosphorylation FEBS Letters. ,vol. 453, pp. 107- 112 ,(1999) , 10.1016/S0014-5793(99)00693-6