THE ROLE OF ANGIOTENSIN II IN THE CONTROL OF HYPOVOLAEMIC THIRST AND SODIUM APPETITE

摘要: Publisher Summary Hypovolemia, or mimicking its effect by reducing the venous return to heart obstructing inferior vena cava, causes increased thirst and, after a delay of some hours, sodium appetite. These two behavioral responses, together with mobilization fluid from tissues and gut, reduction in urine flow, redistribution cardiac output, enable body withstand correct hypovolemia. This chapter reviews that significant positive correlations between drinking fall central pressure arterial suggest stretch receptors circulation are involved hypovolemic thirst. Afferent impulses volume baroreceptors inhibitory Besides being behavior, also influence secretion renin through their on sympathetic tone kidney. The possible involvement renin–angiotensin system is examined chapter, since itself absence changes receptor stimulation.