Zinc metabolism in alcoholic liver disease.

摘要: Alterations in zinc metabolism or deficiency frequently occur patients with alcoholic liver disease. Potential manifestations of include skin lesions, hypogonadism, impaired night vision, immune function, anorexia, altered protein metabolism, diarrhea, and depressed mental function. Because the variety ways which may present disease, clinicians must maintain a high index suspicion for this nutrient when caring these patients. Not only but there also be metabolism. Recent data from hepatitis demonstrate increased serum levels monokine interleukin 1, is known to cause hypozincemia an internal redistribution zinc. This has host metabolic functions other than its effect on mineral that have relevance disease such as fever production, neutrophilia, muscle catabolism. We suggest patient problems either potential implications are discussed.