作者: G. Marchetti , L. Merlo , V. Noseda , E. Nobile
DOI: 10.1007/BF00586769
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摘要: Previous investigations have demonstrated that 60–70% of myocardial venous blood returns from the coronary sinus and this percentage increases by experimentally raising right ventricle systolic pressure. The experiments described were performed in anesthetized open chest dogs showed increase outflow is not due to a change distribution between deep system, but secondary increased left artery inflow. A possible explanation might be intramyocardial ventricular pressure brings about some release certain catecholamines which may induce persisting Beta-blockade can effect prevent any flow arising overdistension ventricle. O2 content (returning mainly ventricle) constantly lower than anterior cardiac Thebesius veins (flowing out ventricle). This difference very probably result mechanical factors play an active role regulating flow. As well known, total resistances are sum two components i.e., a) extravascular compression during systole b) vasomotor tone bed. Vasomotility arteries virtually identical, whereas considerably side heart left. Right therefore less impaired artery; ratio extraction should thus better