Smoking and Endothelial Progenitor Cells: A Revision of Literature
作者： Rossella Di Stefano , Maria Chiara Barsotti , Francesca Felice , Angela Magera , John Lekakis
DOI: 10.2174/138161210792062939
关键词:
摘要: Accumulating evidence indicates that circulating endothelial progenitor cells (EPCs) derived from bone marrow contribute to reendothelialization of injured vessels as well neo-vascularization ischemic lesions in either a direct or an indirect way. Moreover, the number and/or functional activity EPCs are inversely correlated with risk factors for cardiovascular disease. Among different factors, cigarette smoking is major cause reducing numbers and function EPCs. This review revision recent literature on EPC alteration associated smoking. In particular, we show observation effects active second hand smoke (SHS) exposure activity. also considers nicotine other compounds activity, vitro vivo models.
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