Cardiac output during ritodrine treatment in premature labor

摘要: Abstract Unlike other β-mimetic drugs, ritodrine hydrochloride is able to inhibit uterine contractility in premature labor without producing hypotension. Based on previous studies, we have postulated that this homeostatic effect of caused by an increase cardiac output. In 5 women labor, the indicator dilution technique was used check Arterial pressure, amniotic and heart rate were recorded electronically. The systolic blood pressure increased 12 per cent during infusion, while diastolic decreased 10 cent. mean remained stable, but pulse significantly at 30 minutes infusion. A consistent output found which reached highest value, 56 over control, 40 minutes. There a slight decrease 35 above control 60 because reduced venous return attributed inhibition contractility. significance these findings discussed.