Postinfarction ventricular aneurysms.

作者: Brian M. Friedman , Marvin I. Dunn

DOI: 10.1002/CLC.4960180905

关键词:

摘要: Ventricular aneurysms are circumscribed, thin-walled fibrous, noncontractile outpouchings of the ventricle. The majority apically located, true left ventricle (LV) that occur as a consequence transmural myocardial infarction (MI). precursor aneurysm formation appears to be infarct expansion early after acute MI and occurrence generally relates size. presence underlying hypertension use steroids nonsteroidal antiinflammatory agents may promote formation. clinical sequelae include congestive heart failure (CHF), thromboembolism, angina pectoris, ventricular tachyarrhythmias. Late rupture is particular complication false in which pericardium wall. diagnosis suspected by finding diffuse, pansystolic apical thrust, persistent ST-segment elevation on electrocardiogram, distortion cardiac silhouette chest x-ray. This can confirmed using echocardiography, radionuclide ventriculography, catheterization. latter has additional advantage being able delineate coronary anatomy. Management involves prevention, specific therapy for various manifestations, surgery. Therapeutic interventions with thrombolytic agents, aspirin, heparin, beta blockers applied evolution an limit size, thereby reducing tendency toward Patients mild CHF usually controlled standard combination angiotensin-converting enzyme inhibitors, diuretics, digoxin. Thromboembolism best prevented anticoagulation warfarin at least 3 months MI. choice pharmacotherapy tachyarrhythmias should guided electrophysiologic studies. treatment patients pectoris utilizes conventional therapeutic modalities.(ABSTRACT TRUNCATED AT 250 WORDS)

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