Sac1, a putative regulator that is critical for survival of Chlamydomonas reinhardtii during sulfur deprivation.

摘要: The sac1 mutant of Chlamydomonas reinhardtii is aberrant in most the normal responses to sulfur limitation; it cannot synthesize arylsulfatase, does not take up sulfate as rapidly wild-type cells, and periplasmic proteins that normally accumulate during sulfur-limited growth. Here, we show dies much more than cells deprivation; this emphasizes vital role acclimation process. loss viability deprivation only observed light mostly inhibited by DCMU. During sulfur-stress, but mutant, downregulate photosynthesis. Thus, death probably a consequence its inability Furthermore, since SAC1 necessary for downregulation photosynthesis, process must be highly controlled simply result general decrease protein synthesis due limitation. Genomic cDNA copies gene have been cloned. deduced amino acid sequence Sac1 similar an Escherichia coli may involved response E.coli nutrient deprivation.