The locus Om, responsible for the DDK syndrome, maps close to Sigje on mouse chromosome 11.
作者： Patricia A. Baldacci , V�ronique Richoux , Jean-Paul Renard , Jean-Louis Gu�net , Charles Babinet
DOI: 10.1007/BF00353857
关键词: Inbred strain 、 Backcrossing 、 Embryo 、 Blastocyst 、 Locus (genetics) 、 Genetic determinism 、 Biology 、 Genetics 、 Embryonic stem cell 、 Genome
摘要: The DDK inbred strain of mouse has a striking particularity: when females are crossed to males other strains they exhibit reduced fertility, whereas the reciprocal crosses (non-DDK x males) fertile (Wakasugi et al. 1967; Wakasugi 1973). low fertility results from an early embryonic lethality, F1 embryos dying near late morula-early blastocyst stage. Genetic analyses 1974) and nuclear cytoplasmic transfers (Renard Babinet 1986; 1990; Mann 1986), have shown that failure develop is due incompatibility between maternally encoded product non-DDK paternal genome. In order elucidate genetic determinism this we analyzed male progeny backcross BALB/c (BALB/c DDK)F1 set recombinant (RI) strains, established progenitors, mated with females. Our indicate single locus, Om, responsible for syndrome located on Chromosome (Chr) 11, very close Sigje locus.
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